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Emerging evidence indicates that riboflavin deficiency skews microglia toward a pro-inflammatory M1 phenotype, characterized by elevated iNOS, IL-1, and TNF-, whereas riboflavin supplementation promotes an anti-inflammatory M2 phenotype, with higher IL-10 and TGF- expression via STAT6 activation (67, 68)

As a result of the interaction of genetic and environmental factors, people with ASD have excessive ROS production, decreased antioxidant capacity, and mitochondrial dysfunction (92, 93)

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J Exp Bot 65(5):12411257 Cairns NG, Pasternak M, Wachter A, Cobbett CS, Meyer AJ (2006) Maturation of Arabidopsis seeds is dependent on glutathione biosynthesis within the embryo

Park and his research team successfully delineated tumors by monitoring fluorescence dye-labeled nanoparticles accumulating in the tumor tissues by targeting tumor-specific leaky blood vessels